NDLI: Impact of Estradiol Benzoate and Progesterone on the Activity of Carbonic Anhydrase of Erythrocytes in Ducks (Anatis domesticae Caro)

Content Provider	IEEE Xplore Digital Library
Author	Wu, Y.Y. Zhao, X.Z. Li, P.P.
Copyright Year	2010
Abstract	The impact of estradiol benzoate and progesterone on the duck erythrocyte carbonic anhydrase (CA) activity of in vitro culture was studied. The impact of estradiol benzoate and progesterone on CA activity of in vitro culture duck erythrocytes was significant and varied with the incubation durations. When the duck erythrocytes were incubated 2, 14 hours, estradiol benzoate increased the CA activity. When the duck erythrocytes were incubated thirty hours, estradiol benzoate at a higher addition (more than 200 pg ml-1) had no facilitation to the CA activity. Progesterone with addition in the range of 0.5 to 50 ng ml-1 promoted the CA activity when the duck erythrocytes were incubated fourteen hours. The CA activity was also decreased by progesterone when the duck erythrocytes were incubated two hours in MEM medium containing progesterone at the addition of 1 to 10 ng ml-1. Progesterone at the addition of 0.5 ng ml-1 inhibited the CA activity when the duck erythrocytes were incubated thirty hours. These findings can help to understand the interaction between CA and reproductive hormones, and account for the impact of exogenous hormones administration or endogenous estrogens analogs inhalation on the eggshell formation of right thickness and some other biological functions.
Starting Page	1
Ending Page	4
File Size	255767
Page Count	4
File Format	PDF
ISBN	9781424447121
ISSN	21517622
DOI	10.1109/ICBBE.2010.5518031
Language	English
Publisher	Institute of Electrical and Electronics Engineers, Inc. (IEEE)
Publisher Date	2010-06-18
Publisher Place	China
Access Restriction	Subscribed
Rights Holder	Institute of Electrical and Electronics Engineers, Inc. (IEEE)
Subject Keyword	Water Pollution Inhibitors Glands Carbon dioxide Cells (biology) Biochemistry In vitro Zinc Blood
Content Type	Text
Resource Type	Article

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