NDLI: Oxidative stress and immunotoxic effects of lead and their amelioration with myrrh (Commiphora molmol) emulsion.

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Year: 2010 Volume: 48 Issue: Supplementary 1

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Oxidative stress and immunotoxic effects of lead and their amelioration with myrrh (Commiphora molmol) emulsion.

Content Provider	World Health Organization (WHO)-Global Index Medicus
Author	Ashry, Khaled M. El-Sayed, Yasser S. Khamiss, Rania M. El-Ashmawy, Ibrahim M.
Description	Country affiliation: Egypt Author Affiliation: Ashry KM ( Department of Veterinary Forensic Medicine and Toxicology, Faculty of Veterinary Medicine, Alexandria University, Edfina, Rossetta-line, Behera Province, Egypt.)
Abstract	The possible role of Commiphora molmol emulsion (CME) in protecting against lead (PbAc)-induced hepatotoxicity, oxidative stress and immunotoxicity in rabbits was assessed. Six groups of animals were used: groups I (control) and II (PbAc) were not supplemented with CME. Groups III (CME50) and IV (CME50+PbAc) were administered with CME in a dose rate of 50mg/kg bwt, while groups V (CME100) and VI (CME100+PbAc) were received 100mg CME/kg bwt daily p.o for successive 14 weeks. Groups II, IV and VI were given 80 mg PbAc/kg bwt/day orally for 6 weeks starting from the 9th week. At the 12th week, animals were subjected to immunization by a single dose of sheep RBCs. The PbAc-group showed 220% increase in hepatic malondialdehyde levels, while glutathione, glutathione s-transferase and glutathione peroxidase levels decreased. Lead-acetate induced hypoproteinemia and hypoalbuminemia, and increased aminotransferases activity. It reduced the values of lymphocyte transformation test, phagocytic activity, phagocytic index and antibody titer against sheep SRBCs. Interestingly, pretreatment with CME attenuated these adverse effects in a dose-dependent protection. CME, therefore, is a potent antioxidant, and can protect against PbAc-induced hepatic oxidative damage and immunotoxicity by reducing lipid peroxidation and enhancing the antioxidant and immune defense mechanisms.
File Format	HTM / HTML
ISSN	02786915
Issue Number	1
Volume Number	48
e-ISSN	18736351
Journal	Food and Chemical Toxicology
Language	English
Publisher	Elsevier
Publisher Date	2010-01-01
Publisher Place	Great Britain (UK)
Access Restriction	One Nation One Subscription (ONOS)
Subject Keyword	Discipline Toxicology__semicolon__nutritional Discipline Sciences Antioxidants Pharmacology Commiphora Chemistry Immunity Drug Effects Lead Poisoning Immunology Metabolism Oxidative Stress Terpenes Animals Antibody Formation Emulsions Immunity, Humoral Indicators And Reagents Prevention & Control Lipid Peroxidation Liver Pathology Male Phagocytosis Rabbits Journal Article
Content Type	Text
Resource Type	Article
Subject	Medicine Toxicology Food Science

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