NDLI: A review of immune transfer by the placenta.

Please wait, while we are loading the content...

Intravaginal infection with herpes simplex virus type-2 (HSV-2) generates a functional effector memory T cell population that persists in the murine genital tract.

Expression and role of interleukin-23 in human endometrium throughout the menstrual cycle and early pregnancy.

Uterine natural killer cells in peri-implantation endometrium from women with repeated implantation failure after IVF.

Peripheral blood invariant natural killer T cells throughout pregnancy and in preeclamptic women.

Functional analysis of the mannose-binding lectin complement pathway in normal pregnancy and preeclampsia.

A review of immune transfer by the placenta.

Interplay of cytokine polymorphisms and bacterial vaginosis in the etiology of preterm delivery.

Plasma from preeclamptic women activates endothelial cells via monocyte activation in vitro.

Year: 2010 Volume: 86

Year: 2010 Volume: 85

Year: 2010 Volume: 84

Year: 2009 Volume: 83

Year: 2009 Volume: 82

Year: 2009 Volume: 81

Year: 2009 Volume: 80

Year: 2009 Volume: 79

Year: 2008 Volume: 79

Year: 2008 Volume: 78

Year: 2008 Volume: 77

Year: 2007 Volume: 76

Year: 2005 Volume: 67

A review of immune transfer by the placenta.

Content Provider	World Health Organization (WHO)-Global Index Medicus
Author	Chucri, T. M. Monteiro, J. M. Lima, A. R. Salvadori, M. L. B. Kfoury, J. R. Miglino, M. A.
Description	Country affiliation: Brazil Author Affiliation: Chucri TM ( Department of Surgery, College of Veterinary Medicine, University of São Paulo - SP, Brazil. thaischucri@usp.br)
Abstract	Feto-maternal immune transfer occurs via both the placenta in utero and colostrum after birth. The layers between the maternal and fetal circulation systems, known as the placental barrier, regulate immune transfer to the fetus via the placenta. The placental barrier, as well as the type of placental structure, is species specific. The extent of transfer of antibodies from mother to fetus is related to the number of placental barrier layers. Passive immunity via the colostrum is essential in species in which the type of placentation impedes contact between maternal and fetal circulation systems, hindering the transfer of antibodies. In these species, susceptibility to neonatal infections is increased if colostrum is not ingested. Acquired antibodies are of extreme importance for adaptation of the neonate to the extrauterine environment. Based on the aforementioned factors, it was observed that in synepitheliochorial and epitheliochorial placentas immune transfer via the placenta is not possible, except in cases of placental alteration (e.g., placentitis). On the other hand, the mechanism of transfer in endothelial and hemochorial placentas is facilitated compared with other placentas. We conclude that there are no appreciable qualitative differences between the two mechanisms of transfer (placenta and colostrum) and that immune protection in the neonate can be attained by either mechanism.
File Format	HTM / HTML
ISSN	01650378
Issue Number	1-2
Volume Number	87
e-ISSN	18727603
Journal	Journal of Reproductive Immunology
Language	English
Publisher	Elsevier
Publisher Date	2010-12-01
Publisher Place	Ireland
Access Restriction	One Nation One Subscription (ONOS)
Subject Keyword	Discipline Immunology Discipline Reproductive Medicine Colostrum Immunology Fetus Immunity, Maternally-acquired Placenta Placental Circulation Animals Female Humans Physiology Placentation Pregnancy Journal Article Research Support, Non-u.s. Gov't Review
Content Type	Text
Resource Type	Article
Subject	Immunology and Allergy Immunology Reproductive Medicine Obstetrics and Gynecology

Sl.	Authority	Responsibilities	Communication Details
1	Ministry of Education (GoI), Department of Higher Education	Sanctioning Authority	https://www.education.gov.in/ict-initiatives
2	Indian Institute of Technology Kharagpur	Host Institute of the Project: The host institute of the project is responsible for providing infrastructure support and hosting the project	https://www.iitkgp.ac.in
3	National Digital Library of India Office, Indian Institute of Technology Kharagpur	The administrative and infrastructural headquarters of the project	Dr. B. Sutradhar bsutra@ndl.gov.in
4	Project PI / Joint PI	Principal Investigator and Joint Principal Investigators of the project	Dr. B. Sutradhar bsutra@ndl.gov.in Prof. Saswat Chakrabarti will be added soon
5	Website/Portal (Helpdesk)	Queries regarding NDLI and its services	support@ndl.gov.in
6	Contents and Copyright Issues	Queries related to content curation and copyright issues	content@ndl.gov.in
7	National Digital Library of India Club (NDLI Club)	Queries related to NDLI Club formation, support, user awareness program, seminar/symposium, collaboration, social media, promotion, and outreach	clubsupport@ndl.gov.in
8	Digital Preservation Centre (DPC)	Assistance with digitizing and archiving copyright-free printed books	dpc@ndl.gov.in
9	IDR Setup or Support	Queries related to establishment and support of Institutional Digital Repository (IDR) and IDR workshops	idr@ndl.gov.in

Role of IgG antibodies in association with placental function and immunologic diseases in human pregnancy.

Pregnancy and multiple sclerosis: feto-maternal immune cross talk and its implications for disease activity.

MicroRNAs in pregnancy.

Galectins in angiogenesis: consequences for gestation.

Foetal immune programming: hormones, cytokines, microbes and regulatory T cells.

The role of the Nod-like receptor family in trophoblast innate immune responses.

Maternal-fetal interactions, predictive markers for preeclampsia, and programming.

Deep placentation.

Maternal microchimerism: lessons learned from murine models.

A review of immune transfer by the placenta.

Similar Documents

Role of IgG antibodies in association with placental function and immunologic diseases in human pregnancy.

Pregnancy and multiple sclerosis: feto-maternal immune cross talk and its implications for disease activity.

MicroRNAs in pregnancy.

Galectins in angiogenesis: consequences for gestation.

Foetal immune programming: hormones, cytokines, microbes and regulatory T cells.

The role of the Nod-like receptor family in trophoblast innate immune responses.

Maternal-fetal interactions, predictive markers for preeclampsia, and programming.

Deep placentation.

Maternal microchimerism: lessons learned from murine models.

A review of immune transfer by the placenta.