NDLI: Inhibition of human immunodeficiency virus type 1 production in infected peripheral blood mononuclear cells by human leukocyte antigen class I-specific antibodies: evidence for a novel antiviral mechanism.

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The concentration of Ca2+ that solubilizes outer capsid proteins from rotavirus particles is dependent on the strain.

Frequency of multiple Epstein-Barr virus infections in T-cell-immunocompromised individuals.

Isolation of intertypic recombinants of Epstein-Barr virus from T-cell-immunocompromised individuals.

The adenovirus E3-14.7K protein and the E3-10.4K/14.5K complex of proteins, which independently inhibit tumor necrosis factor (TNF)-induced apoptosis, also independently inhibit TNF-induced release of arachidonic acid.

AP1 enhances polyomavirus DNA replication by promoting T-antigen-mediated unwinding of DNA.

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Self-deleting retrovirus vectors for gene therapy.

Serial passage of tobacco rattle virus under different selection conditions results in deletion of structural and nonstructural genes in RNA 2.

Detection of simian immunodeficiency virus (SIV)-specific CD8+ T cells in macaques protected from SIV challenge by prior SIV subunit vaccination.

Mengovirus leader is involved in the inhibition of host cell protein synthesis.

Human immunodeficiency virus type 1 gp120 induces anergy in human peripheral blood lymphocytes by inducing interleukin-10 production.

Unintegrated bovine leukemia virus DNA: association with viral expression and disease.

Inactivation of murine leukemia virus by compounds that react with the zinc finger in the viral nucleocapsid protein.

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Identification and characterization of a hepatitis delta virus RNA transcriptional promoter.

Human immunodeficiency virus Type 1 nucleocapsid protein (NCp7) directs specific initiation of minus-strand DNA synthesis primed by human tRNA(Lys3) in vitro: studies of viral RNA molecules mutated in regions that flank the primer binding site.

Down-regulation of paramyxovirus hemagglutinin-neuraminidase glycoprotein surface expression by a mutant fusion protein containing a retention signal for the endoplasmic reticulum.

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cis-active structural motifs involved in specific encapsidation of Moloney murine leukemia virus RNA.

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Structural analysis of human immunodeficiency virus type 1 Gag protein interactions, using cysteine-specific reagents.

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Interaction between hepatitis C virus core protein and E1 envelope protein.

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The tax gene of human T-cell leukemia virus type 2 is essential for transformation of human T lymphocytes.

Antisense oligonucleotide inhibition of hepatitis C virus gene expression in transformed hepatocytes.

Inhibition of human immunodeficiency virus type 1 production in infected peripheral blood mononuclear cells by human leukocyte antigen class I-specific antibodies: evidence for a novel antiviral mechanism.

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Sequence and structural determinants required for priming of plus-strand DNA synthesis by the human immunodeficiency virus type 1 polypurine tract.

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Characterization of the functional properties of env genes from long-term survivors of human immunodeficiency virus type 1 infection.

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Paradoxical interactions between human delta hepatitis agent RNA and the cellular protein kinase PKR.

Transcriptional activation of a retrovirus enhancer by CBF (AML1) requires a second factor: evidence for cooperativity with c-Myb.

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Inhibition of human immunodeficiency virus type 1 production in infected peripheral blood mononuclear cells by human leukocyte antigen class I-specific antibodies: evidence for a novel antiviral mechanism.

Content Provider	PubMed Central
Author	Briant, L. Benkirane, M. Girard, M. Hirn, M. Iosef, C. Devaux, C.
Abstract	A well-characterized mechanism by which anti-HLA class I monoclonal antibodies (MAb) inhibit human immunodeficiency virus type 1 (HIV-1) propagation in in vitro cell cultures is the neutralization of the virus through interactions with HLA molecules associated with the virion envelope. Yet, the possibility that another mechanism of inhibition might affect a postbinding stage of the virus life cycle has been strongly suggested by our previous investigations. To demonstrate that the interaction of MAb B1-1G6 with the light chain of cell surface-expressed HLA class I molecules inhibits a postbinding step of the HIV-1 life cycle, peripheral blood mononuclear cells (PBMCs) were exposed to viruses grown in HLA class I-negative, CD4-positive cells (these viruses, which did not carry HLA class I molecules, cannot be neutralized by anti-HLA MAb during the first round of infection), and PCR was used at various times postexposure to search for the different forms of HIV-1 DNA and RNA in virus-exposed PBMCs cultured in either the presence or [correction of] absence of MAb B1-1G6. Although viral DNA was found in MAb B1-1G6-treated cells, spliced HIV-1 mRNA could not be detected in those cells. In contrast, HIV-1 gene expression was found in HIV-1-infected PBMCs treated with B9-12-1, another HLA class I-specific MAb which prevents infection of cells by cell-free viruses but which fails to inhibit cell-to-cell transmission of HIV-1. These results highlight a second antiviral mechanism by which anti-HLA MAb inhibit in vitro HIV-1 propagation.
Starting Page	5213
File Format	PDF
ISSN	10985514
e-ISSN	10985514
Journal	Journal of Virology
Issue Number	8
Volume Number	70
Language	English
Publisher Date	1996-08-01
Access Restriction	Open
Subject Keyword	Research in Higher Education
Content Type	Text
Resource Type	Article
Subject	Virology Immunology Microbiology Insect Science

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