NDLI: Kinase signaling in the spindle checkpoint.

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Kinase signaling in the spindle checkpoint.

Content Provider	World Health Organization (WHO)-Global Index Medicus
Author	Kang, Jungseog Yu, Hongtao
Description	Author Affiliation: Kang J ( Department of Pharmacology, Howard Hughes Medical Institute, University of Texas Southwestern Medical Center, Dallas, Texas 75390, USA.)
Abstract	The spindle checkpoint is a cell cycle surveillance system that ensures the fidelity of chromosome segregation. In mitosis, it elicits the 'wait anaphase' signal to inhibit the anaphase-promoting complex or cyclosome until all chromosomes achieve bipolar microtubule attachment and align at the metaphase plate. Because a single kinetochore unattached to microtubules activates the checkpoint, the wait anaphase signal is thought to be generated by this kinetochore and is then amplified and distributed throughout the cell to inhibit the anaphase-promoting complex/cyclosome. Several spindle checkpoint kinases participate in the generation and amplification of this signal. Recent studies have begun to reveal the activation mechanisms of these checkpoint kinases. Increasing evidence also indicates that the checkpoint kinases not only help to generate the wait anaphase signal but also actively correct kinetochore-microtubule attachment defects.
ISSN	00219258
e-ISSN	1083351X
Journal	Journal of Biological Chemistry
Issue Number	23
Volume Number	284
Language	English
Publisher	American Society for Biochemistry and Molecular Biology (United States)
Publisher Date	2009-06-05
Publisher Place	United States
Access Restriction	Open
Subject Keyword	Chromosome Segregation Mitogen-Activated Protein Kinases Metabolism Spindle Apparatus Aneuploidy Animals Aurora Kinases Cell Cycle Proteins Centromere Enzymology Enzyme Activation Kinetochores Microtubules Genetics Mitosis Neoplasms Protein Binding Protein-Serine-Threonine Kinases Xenopus Biochemistry Molecular Biology
Content Type	Text
Resource Type	Article
Subject	Cell Biology Biochemistry Molecular Biology

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