NDLI: Exposure, epidemiology, and mechanism of the environmental toxicant manganese.

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Long-term persistence of seeded grass species: an unwanted side effect of ecological restoration.

In search for a compromise between biodiversity conservation and human health protection in restoration of fly ash deposits: effect of anti-dust treatments on five groups of arthropods.

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Assessment of heavy metal contamination levels and toxicity in sediments and fishes from the Mediterranean Sea (southern coast of Sfax, Tunisia).

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A comprehensive assessment of agricultural intensification scenarios for the Dongting Lake basin in south-central China in 2030.

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Exposure, epidemiology, and mechanism of the environmental toxicant manganese.

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F sorption/desorption on two soils and on different by-products and waste materials.

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Exposure, epidemiology, and mechanism of the environmental toxicant manganese.

Content Provider	World Health Organization (WHO)-Global Index Medicus
Author	Culbreth, Megan Aschner, Michael Chen, Pan
Description	Country affiliation: United States Author Affiliation: Chen P ( Department of Molecular Pharmacology, Albert Einstein College of Medicine, Forchheimer Building, 1300 Morris Park Avenue, Bronx, NY, 10461, USA.); Culbreth M ( Department of Molecular Pharmacology, Albert Einstein College of Medicine, Forchheimer Building, 1300 Morris Park Avenue, Bronx, NY, 10461, USA.); Aschner M ( Department of Molecular Pharmacology, Albert Einstein College of Medicine, Forchheimer Building, 1300 Morris Park Avenue, Bronx, NY, 10461, USA. michael.aschner@einstein.yu.edu.)
Abstract	It has become increasingly apparent that global manganese (Mn) pollution to air and water is a significant threat to human health. Despite this recognition, research is only beginning to comprehend the detrimental effects of exposure. Mn, while essential, is particularly harmful to the central nervous system, and overexposure is symptomatic of several neurological disorders. At-risk populations have been identified, but it is still unclear whether typical exposure levels have any long-term consequences. Those at an elevated risk have diminished intellectual function, learning and memory, and mental development. While the overall mechanism of toxicity is undetermined, Mn has been found to induce oxidative stress, exacerbate mitochondrial dysfunction, dysregulate autophagy, and promote apoptosis, ultimately enhancing neurodegeneration. Extrapolation of this in vitro and in vivo data to humans is difficult. There is a definite need to correlate epidemiological studies with causative effects. It is imperative that research efforts endure, so threats are appropriately identified and exposure properly regulated.
ISSN	09441344
Issue Number	14
Journal	Environmental Science and Pollution Research
Volume Number	23
e-ISSN	16147499
Language	English
Publisher	Springer
Publisher Date	2016-07-01
Publisher Place	Germany
Access Restriction	One Nation One Subscription (ONOS)
Subject Keyword	Discipline Environmental Science Discipline Environmental Chemistry
Content Type	Text
Resource Type	Article
Subject	Environmental Chemistry Health, Toxicology and Mutagenesis Pollution Medicine

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