NDLI: Model for capping derived from inhibition of surface receptor capping by free fatty acids.

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Model for capping derived from inhibition of surface receptor capping by free fatty acids.

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Model for capping derived from inhibition of surface receptor capping by free fatty acids.

Content Provider	World Health Organization (WHO)-Global Index Medicus
Author	Karnovsky, M. J. Dragsten, P. Klausner, R. D. Bhalla, D. K. Hoover, R. L.
Abstract	When low concentrations (2-5 mole %) of cis unsaturated free fatty acids (group A) are intercalated into lymphocyte plasma membrane, capping is inhibited. No effect is seen with trans unsaturated or saturated fatty acids (group B). The capping inhibition is reversible with increasing doses of extracellular calcium. Fluorescence photobleaching recovery has shown that the group A free fatty acids do not inhibit the receptor immobilization associated with patch formation, but inhibit the final energy-dependent movement of the patched receptors into a cap. We have also shown that the group A free fatty acids cause a shift in membrane-bound calcium to the lipid phase from probable protein-associated sites. We have incorporated these findings into a model for capping and membrane-cytoskeletal interactions.
ISSN	00278424
e-ISSN	10916490
Journal	Proceedings of the National Academy of Sciences of the United States of America
Issue Number	1
Volume Number	77
Language	English
Publisher	National Academy of Sciences
Publisher Date	1980-05-01
Publisher Place	United States
Access Restriction	Open
Subject Keyword	Fatty Acids, Nonesterified Pharmacology Immunologic Capping Drug Effects Lymphocytes Immunology Receptors, Antigen, B-Cell Metabolism Animals Calcium Cell Membrane Physiology Cytoskeleton Diffusion Fatty Acids, Unsaturated Ultrastructure Mice Models, Theoretical Research Support, U.S. Gov't, P.H.S. Multidisciplinary
Content Type	Text
Resource Type	Article
Subject	Multidisciplinary

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