NDLI: Zebrafish and cardiac toxicology

Please wait, while we are loading the content...

Cardiovascular Toxicology : Volume 17

Cardiovascular Toxicology : Volume 16

Cardiovascular Toxicology : Volume 15

Cardiovascular Toxicology : Volume 14

Cardiovascular Toxicology : Volume 13

Cardiovascular Toxicology : Volume 12

Cardiovascular Toxicology : Volume 11

Cardiovascular Toxicology : Volume 10

Cardiovascular Toxicology : Volume 9

Cardiovascular Toxicology : Volume 8

Cardiovascular Toxicology : Volume 7

Cardiovascular Toxicology : Volume 6

Cardiovascular Toxicology : Volume 5

Cardiovascular Toxicology : Volume 5, Issue 4, December 2005

Cardiovascular Toxicology : Volume 5, Issue 3, September 2005

Cardiovascular Toxicology : Volume 5, Issue 2, June 2005

Environmental cardiovascular disease

The transcribed genome and the heritable basis of essential hypertension

A gene-environment interaction model of stress-induced hypertension

Hypertension and SNP genotyping in antihypertensive treatment

Atherosclerosis : From genetic polymorphisms to system genetics

Modification of environmental toxicity by nutrients : Implications in atherosclerosis

The role of phospholipases in lipid modification and atherosclerosis

NF-κB in cardiovascular disease : Diverse and specific effects of a “General” transcription factor?

Zebrafish and cardiac toxicology

Inhibition of neovascularization by environmental agents

Characterization of spontaneous and chemically induced cardiac lesions in rodent model systems : The national toxicology program experience

Cardiovascular Toxicology : Volume 5, Issue 1, March 2005

Cardiovascular Toxicology : Volume 4

Cardiovascular Toxicology : Volume 3

Cardiovascular Toxicology : Volume 2

Cardiovascular Toxicology : Volume 1

Zebrafish and cardiac toxicology

Content Provider	Springer Nature Link
Author	Heideman, Warren Antkiewicz, Dagmara S. Carney, Sara A. Peterson, Richard E.
Copyright Year	2005
Abstract	Model systems are a mainstay in toxicological research. Zebrafish are rapidly becoming an important model organism for studying vertebrate development. The advantages of zebrafish: short reproductive cycle, production of numerous transparent, synchronously developing embryos, low cost, and standardization make zebrafish and attractive model for toxicologists as well. The use of these fish to study heart development has moved forward very rapidly, laying the groundwork for studying the effects of chemicals on cardiac development and function. Here we describe approaches that can be used to study cardiac toxicity in developing zebrafish, focusing on examples where zebrafish embryos have been especially useful in understanding the impact of specific toxicants on heart development and function.
Starting Page	203
Ending Page	214
Page Count	12
File Format	PDF
ISSN	15307905
Journal	Cardiovascular Toxicology
Volume Number	5
Issue Number	2
e-ISSN	15590259
Language	English
Publisher	Humana Press
Publisher Date	2005-01-01
Publisher Place	Totowa
Access Restriction	One Nation One Subscription (ONOS)
Subject Keyword	2,3,7,8-tetrachlorodibenzo-p-dioxin TCDD toxicity zebrafish heart development Pharmacology/Toxicology Cardiology
Content Type	Text
Resource Type	Article
Subject	Molecular Biology Toxicology Cardiology and Cardiovascular Medicine

Sl.	Authority	Responsibilities	Communication Details
1	Ministry of Education (GoI), Department of Higher Education	Sanctioning Authority	https://www.education.gov.in/ict-initiatives
2	Indian Institute of Technology Kharagpur	Host Institute of the Project: The host institute of the project is responsible for providing infrastructure support and hosting the project	https://www.iitkgp.ac.in
3	National Digital Library of India Office, Indian Institute of Technology Kharagpur	The administrative and infrastructural headquarters of the project	Dr. B. Sutradhar bsutra@ndl.gov.in
4	Project PI / Joint PI	Principal Investigator and Joint Principal Investigators of the project	Dr. B. Sutradhar bsutra@ndl.gov.in Prof. Saswat Chakrabarti will be added soon
5	Website/Portal (Helpdesk)	Queries regarding NDLI and its services	support@ndl.gov.in
6	Contents and Copyright Issues	Queries related to content curation and copyright issues	content@ndl.gov.in
7	National Digital Library of India Club (NDLI Club)	Queries related to NDLI Club formation, support, user awareness program, seminar/symposium, collaboration, social media, promotion, and outreach	clubsupport@ndl.gov.in
8	Digital Preservation Centre (DPC)	Assistance with digitizing and archiving copyright-free printed books	dpc@ndl.gov.in
9	IDR Setup or Support	Queries related to establishment and support of Institutional Digital Repository (IDR) and IDR workshops	idr@ndl.gov.in

Altering HIF-1α Through 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) Exposure Affects Coronary Vessel Development

Perinatal 2,3,7,8-Tetrachlorodibenzo-p-dioxin Exposure Sensitizes Offspring to Angiotensin II-induced Hypertension

Blocking Expression of AHR2 and ARNT1 in Zebrafish Larvae Protects Against Cardiac Toxicity of 2,3,7,8-Tetrachlorodibenzo-p-dioxin

Hypertension, Cardiac Hypertrophy, and Impaired Vascular Relaxation Induced by 2,3,7,8-Tetrachlorodibenzo-p-Dioxin are Associated with Increased Superoxide

Dioxin (2,3,7,8-tetrachlorodibenzo-p-doxin) enhances triggered afterdepolarizations in rat ventricular myocytes

Interactions between 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) and Hypoxia Signaling Pathways in Zebrafish: Hypoxia Decreases Responses to TCDD in Zebrafish Embryos

2,3,7,8 Tetrachlorodibenzo-p-dioxin-induced RNA abundance changes identify Ackr3, Col18a1, Cyb5a and Glud1 as candidate mediators of toxicity

Altering HIF-1α Through 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) Exposure Affects Coronary Vessel Development

2,3,7,8-Tetrachlorodibenzo-p-dioxin Toxicity in the Zebrafish Embryo: Altered Regional Blood Flow and Impaired Lower Jaw Development

Zebrafish and cardiac toxicology

Similar Documents

Altering HIF-1α Through 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) Exposure Affects Coronary Vessel Development

Perinatal 2,3,7,8-Tetrachlorodibenzo-p-dioxin Exposure Sensitizes Offspring to Angiotensin II-induced Hypertension

Blocking Expression of AHR2 and ARNT1 in Zebrafish Larvae Protects Against Cardiac Toxicity of 2,3,7,8-Tetrachlorodibenzo-p-dioxin

Hypertension, Cardiac Hypertrophy, and Impaired Vascular Relaxation Induced by 2,3,7,8-Tetrachlorodibenzo-p-Dioxin are Associated with Increased Superoxide

Dioxin (2,3,7,8-tetrachlorodibenzo-p-doxin) enhances triggered afterdepolarizations in rat ventricular myocytes

Interactions between 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD) and Hypoxia Signaling Pathways in Zebrafish: Hypoxia Decreases Responses to TCDD in Zebrafish Embryos

2,3,7,8 Tetrachlorodibenzo-p-dioxin-induced RNA abundance changes identify Ackr3, Col18a1, Cyb5a and Glud1 as candidate mediators of toxicity

Altering HIF-1α Through 2,3,7,8-Tetrachlorodibenzo-p-Dioxin (TCDD) Exposure Affects Coronary Vessel Development

2,3,7,8-Tetrachlorodibenzo-p-dioxin Toxicity in the Zebrafish Embryo: Altered Regional Blood Flow and Impaired Lower Jaw Development

Zebrafish and cardiac toxicology