NDLI: Severe sepsis and Toll-like receptors

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TLRs in disease

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Toll like receptor-5: protecting the gut from enteric microbes

TLR2 in murine atherosclerosis

Severe sepsis and Toll-like receptors

Role of TLRs/MyD88 in host resistance and pathogenesis during protozoan infection: lessons from malaria

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Severe sepsis and Toll-like receptors

Content Provider	Springer Nature Link
Author	Gao, Hongmei Leaver, Susannah K. Burke Gaffney, Anne Finney, Simon J.
Copyright Year	2007
Abstract	Severe sepsis dominates the mortality of non-cardiac intensive care units. The ingenious Toll-like receptor (TLR) system can recognise many infectious organisms through relatively few receptors to trigger pro-inflammatory and anti-inflammatory cytokine release. Further complexity arises from positive and negative signalling feedback loops. Severe sepsis may be a consequence of an inappropriately excessive response or inadequate endogenous negative feedback. Therapies targeting these pathways are currently being evaluated. Alternatively, in clinical scenarios such as compensatory anti-inflammatory response syndrome, chronic viral sepsis or inadequate vaccine function, TLR signalling may be inadequate. TLR agonists may augment the innate response and are being investigated.
Starting Page	29
Ending Page	40
Page Count	12
File Format	PDF
ISSN	18632297
Journal	Springer Seminars in Immunopathology
Volume Number	30
Issue Number	1
e-ISSN	18632300
Language	English
Publisher	Springer-Verlag
Publisher Date	2007-12-11
Publisher Place	Berlin, Heidelberg
Access Restriction	One Nation One Subscription (ONOS)
Subject Keyword	Sepsis Toll-like receptors Gram-positive bacterial infections Gram-negative bacterial infections Viral infections Fungal infections Internal Medicine Immunology
Content Type	Text
Resource Type	Article
Subject	Immunology and Allergy Immunology

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Update on the role of Toll-like receptors during bacterial infections and sepsis.

Pathogenesis of sepsis: new concepts and implications for future treatment.

Serum procalcitonin levels distinguish Gram-negative bacterial sepsis from Gram-positive bacterial and fungal sepsis

Clinical usefulness of procalcitonin as a marker of sepsis: a novel predictor of causative pathogens?

NOX enzymes and Toll-like receptor signaling

Lipopolysaccharide-Binding Protein Serum Levels in Patients with Severe Sepsis Due to Gram-Positive and Fungal Infections

Infections and the antiphospholipid syndrome.

Severe sepsis and Toll-like receptors

Similar Documents

Toll-like receptors in the skin

Immunity to bacterial infection in the chicken.

Update on the role of Toll-like receptors during bacterial infections and sepsis.

Pathogenesis of sepsis: new concepts and implications for future treatment.

Serum procalcitonin levels distinguish Gram-negative bacterial sepsis from Gram-positive bacterial and fungal sepsis

Clinical usefulness of procalcitonin as a marker of sepsis: a novel predictor of causative pathogens?

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Severe sepsis and Toll-like receptors