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Striatal inhomogeneities and basal ganglia function.
| Content Provider | Semantic Scholar |
|---|---|
| Author | Penney, John B. Young, Anne B. |
| Copyright Year | 1986 |
| Abstract | It has always been something of a mystery as to why different diseases of the basal ganglia may produce completely opposite effects on movement. Thus, defective dopaminergic inhibitory input into the striatum in Parkinson’s disease causes an akinetic-rigid syndrome, while destruction of the striatum in Huntington’s disease usually causes chorea. This theoretic paradox may be resolved by more detailed examination of the exact sites of striatal damage in the two opposing conditions (1). C. D. Marsden |
| Starting Page | 3 |
| Ending Page | 15 |
| Page Count | 13 |
| File Format | PDF HTM / HTML |
| Alternate Webpage(s) | https://deepblue.lib.umich.edu/bitstream/handle/2027.42/50393/870010102_ftp.pdf?isAllowed=y&sequence=1 |
| PubMed reference number | 2848190v1 |
| Volume Number | 1 |
| Issue Number | 1 |
| Journal | Movement disorders : official journal of the Movement Disorder Society |
| Language | English |
| Access Restriction | Open |
| Subject Keyword | Basal Ganglia Chorea Dopamine Hydrochloride Muscle Rigidity Neostriatum Parkinson Disease Parkinsonian Disorders |
| Content Type | Text |
| Resource Type | Article |