NDLI: At the frog neuromuscular junction prostaglandin synthase inhibitors depress and PGE2 partially restores quantal acetylcholine release

Content Provider	Semantic Scholar
Author	Madden, Kathleen Shaver Kloot, William Van Der
Copyright Year	1982
Abstract	Abstract Indomethacin and 5,8,11,14-eicosatetraynoic acid inhibit prostaglandin synthesis. The drugs irreversibly decreased quantal acetylcholine (ACh) release at the frog neuromuscular junction and increased the latency of the stimulated response. Subsequent exposure to poostaglandin E2 increased e.p.p. amplitudes by increasing quantal release and the effectiveness of single quanta. These results resemble the effects of these agents on ACh release at parasympathetic-smooth muscle junctions.
Starting Page	464
Ending Page	468
Page Count	5
File Format	PDF HTM / HTML
DOI	10.1016/0006-8993(82)90888-5
Alternate Webpage(s)	https://api.elsevier.com/content/article/pii/0006899382908885
Alternate Webpage(s)	https://www.sciencedirect.com/science/article/pii/0006899382908885?dgcid=api_sd_search-api-endpoint
PubMed reference number	6277436
Alternate Webpage(s)	https://doi.org/10.1016/0006-8993%2882%2990888-5
Journal	Medline
Volume Number	234
Journal	Brain Research
Language	English
Access Restriction	Open
Content Type	Text
Resource Type	Article

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At the frog neuromuscular junction prostaglandin synthase inhibitors depress and PGE2 partially restores quantal acetylcholine release