NDLI: Helicobacter pylori gastritis, peptic ulcer, and gastric cancer: clinical and molecular aspects.

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Helicobacter pylori gastritis, peptic ulcer, and gastric cancer: clinical and molecular aspects.

Content Provider	Semantic Scholar
Copyright Year	1997
Abstract	Helicobacter pylori causes specific ultrastructural changes to the gastric mucosa. In developing countries a high percentage of infants acquire this infection, which initially causes a transient drop in stomach acid and thus allows transit of lower bowel pathogens, with consequent diarrhea and malnutrition. When infection occurs at an early age, the acid-producing cells of the stomach are involved in the inflammation, and the lifelong reduced acid output means a duodenal ulcer rarely develops. However, lifelong gastric inflammation leads in due course to atrophy, and in the presence of other factors gastric cancer may develop. People infected with H. pylori on average are of shorter stature than uninfected people. Adherence of H. pylori to the gastric mucosa is a prerequisite for infection, and a new binary model of adherence has been shown recently. Chaperonins of H. pylori induce macrophages to secrete cytokines, which leads to an immunologic cascade and inflammation.
File Format	PDF HTM / HTML
Alternate Webpage(s)	https://oup.silverchair-cdn.com/oup/backfile/Content_public/Journal/cid/25/5/10.1086/516077/2/25-5-1017.pdf?Expires=1491354430&Key-Pair-Id=APKAIUCZBIA4LVPAVW3Q&Signature=IYPMSZugbgGU2HM081yYpghFo8xBxWD58RNzAsUDceMUgIfKAy6D5uaSUQLK3uxtWyREr3aBAwOTimhSUTFxqx9esR~eEL-0w-7QiFcuxYjsDnKZWfnddHhV9abrJP8sMMGZ1GmMDZX3tsqTNkUxhuOR5ikchGDYSsbVTt7N9FMMYyT2FPPZjkYYaOZMNw1-RT8BU0yN5rnPELHelBr0W3PETOHrKz76ViXu92MJOuHcxIeVPZizLxxUSVzAW4EcioR83ugZ4DQ1NIqIJsvifCfPeReRXBn6UuBTlrTGuSBi099Wty1vcVUbEYpxmV6ENiQjeKAeC-3f91apl-VvyQ__
PubMed reference number	9402348v1
Volume Number	25
Issue Number	5
Journal	Clinical infectious diseases : an official publication of the Infectious Diseases Society of America
Language	English
Access Restriction	Open
Subject Keyword	Body Height Cascade Device Component Cell secretion Diarrhea Duodenal Ulcer Duodenum Gastric Tissue Gastric mucosa Helicobacter pylori gastritis Intestines Malnutrition Peptic Ulcer Stomach Carcinoma chaperonin
Content Type	Text
Resource Type	Article

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