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| Content Provider | PubMed Central |
|---|---|
| Author | Yana, Kiselyeva Nedellec, Rebecca Ramos, Alejandra Pastore, Cristina Margolis, Leonid B. Mosier, Donald E. |
| Copyright Year | 2007 |
| Abstract | CCR5-using human immunodeficiency virus type 1 (HIV-1) isolates typically gain CXCR4 use via multiple mutations in V3 and often V1/V2 regions of envelope, and patterns of mutations are distinct for each isolate. Here, we report that multiple CXCR4-using variants of a parental CCR5-using HIV-1 isolate, SF162, obtained by either target cell selection or CCR5 inhibition have a common mutation pattern characterized by the same two V3 mutations and that these mutations preexisted in some of the SF162 stocks. These results imply that SF162 has a single pathway for acquiring CXCR4 use and that prolonged culture is sufficient to select for R5X4 variants. |
| Related Links | http://dx.doi.org/10.1128/jvi.02310-06 |
| Ending Page | 3661 |
| Page Count | 5 |
| Starting Page | 3657 |
| File Format | |
| ISSN | 0022538X |
| e-ISSN | 10985514 |
| Journal | Journal of Virology |
| Issue Number | 7 |
| Volume Number | 81 |
| Language | English |
| Publisher | American Society for Microbiology |
| Publisher Date | 2007-04-01 |
| Access Restriction | Open |
| Rights Holder | American Society for Microbiology |
| Subject Keyword | Immunology Virology Research in Higher Education |
| Content Type | Text |
| Resource Type | Article |
| Subject | Virology Immunology Microbiology Insect Science |
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