NDLI: Glatiramer acetate treatment of multiple sclerosis: an immunological perspective.

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In vivo administration of the recombinant IL-7/hepatocyte growth factor ß hybrid cytokine efficiently restores thymopoiesis and naive T cell generation in lethally irradiated mice after syngeneic bone marrow transplantation.

Molecular and functional characterization of mouse S5D-SRCRB: a new group B member of the scavenger receptor cysteine-rich superfamily.

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Proteasome inhibition with bortezomib depletes plasma cells and autoantibodies in experimental autoimmune myasthenia gravis.

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IL-3 attenuates collagen-induced arthritis by modulating the development of Foxp3+ regulatory T cells.

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Essential role of E3 ubiquitin ligase activity in Cbl-b-regulated T cell functions.

Cutting Edge: The murine high-affinity IgG receptor FcÎ³RIV is sufficient for autoantibody-induced arthritis.

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A single nucleotide polymorphism in NF-κB inducing kinase is associated with mortality in septic shock.

Differential expression of NLRP3 among hematopoietic cells.

CD4 and CD8 T cell immune activation during chronic HIV infection: roles of homeostasis, HIV, type I IFN, and IL-7.

Comment on 'Therapeutic targeting of Syk in autoimmune diabetes'.

Glatiramer acetate treatment of multiple sclerosis: an immunological perspective.

Apolipoprotein E and peptide mimetics modulate inflammation by binding the SET protein and activating protein phosphatase 2A.

Retraction: The IL-15R chain signals through association with Syk in human B cells.

Retraction: Mast cells express novel functional IL-15 receptor isoforms.

Retraction: Extracellular ATP induces cytokine expression and apoptosis through P2X7 receptor in murine mast cells.

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Glatiramer acetate treatment of multiple sclerosis: an immunological perspective.

Content Provider	World Health Organization (WHO)-Global Index Medicus
Author	Racke, Michael K. Lovett-Racke, Amy E.
Description	Country affiliation: United States Author Affiliation: Racke MK ( Department of Neurology, The Ohio State University Medical Center, Columbus, OH 43210-1228, USA. michael.racke@osumc.edu)
Abstract	Glatiramer acetate (GA) has been used as an immunomodulatory agent for the treatment of relapsing-remitting multiple sclerosis (MS) in the United States since 1996. It is currently one of two first-line agents for use in the treatment of relapsing-remitting MS. GA was the first agent to be used in the treatment of MS that was developed using the animal model of MS called experimental autoimmune encephalomyelitis. In this commentary, we examine the development of GA as a treatment for MS and discuss its mechanism of action as suggested by recent studies using modern immunologic methods.
ISSN	00221767
e-ISSN	15506606
Journal	The Journal of Immunology
Issue Number	4
Volume Number	186
Language	English
Publisher	The American Association of Immunologists
Publisher Date	2011-02-15
Publisher Place	United States
Access Restriction	Open
Subject Keyword	Encephalomyelitis, Autoimmune, Experimental Multiple Sclerosis, Relapsing-remitting Drug Therapy Immunology Peptides Therapeutic Use Animals Antigen-presenting Cells Drug Effects Pathology Disease Models, Animal Glatiramer Acetate Administration & Dosage T-lymphocytes, Regulatory Discipline Immunology
Content Type	Text
Resource Type	Article
Subject	Immunology and Allergy Immunology

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