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| Content Provider | World Health Organization (WHO)-Global Index Medicus |
|---|---|
| Author | Van Ree, J. M. Plomp, G. J. |
| Abstract | 1 Fragments of the N terminal part of adrenocorticotrophic hormone (ACTH) inhibited the electrically evoked contractions of the mouse vas deferens. This inhibition could be antagonized by naloxone. 2 The same fragments displaced radiolabelled morphine from morphine antiserum. 3 Structure-activity relationship studies showed that in both assay systems the active core is located within the sequence ACTH 7--10. 4 It is postulated that the Trp9 residue and the peptide bond between Trp9 and Gly10 are particularly important for interaction of ACTH fragments with morphine receptors. |
| ISSN | 00071188 |
| e-ISSN | 14765381 |
| Journal | British Journal of Pharmacology |
| Issue Number | 2 |
| Volume Number | 64 |
| Language | English |
| Publisher | Wiley Online Library(on behalf of The British Pharmacological Society) |
| Publisher Date | 1978-10-01 |
| Publisher Place | Great Britain (UK) |
| Access Restriction | Open |
| Subject Keyword | Adrenocorticotropic Hormone Analogs & Derivatives Morphine Pharmacology Muscle Contraction Drug Effects Muscle, Smooth Animals Antibodies Binding, Competitive In Vitro Techniques Mice Immunology Peptide Fragments Structure-Activity Relationship Vas Deferens |
| Content Type | Text |
| Resource Type | Article |
| Subject | Pharmacology |
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