NDLI: Eosinophilic vasculitis: time for recognition of a new entity?

Content Provider	World Health Organization (WHO)-Global Index Medicus
Author	Law, Arjun Datt Varma, Subhash Varma, Neelam Khadwal, Alka Prakash, Gaurav Suri, Vikas Kumari, Savita Malhotra, Pankaj
Description	Author Affiliation: Law AD ( Department of Internal Medicine, Postgraduate Institute of Medical Education and Research (PGIMER), Chandigarh, 160 012 India.); Varma S ( Department of Internal Medicine, Postgraduate Institute of Medical Education and Research (PGIMER), Chandigarh, 160 012 India.); Varma N ( Department of Haematology, Postgraduate Institute of Medical Education and Research (PGIMER), Chandigarh, 160 012 India.); Khadwal A ( Department of Internal Medicine, Postgraduate Institute of Medical Education and Research (PGIMER), Chandigarh, 160 012 India.); Prakash G ( Department of Internal Medicine, Postgraduate Institute of Medical Education and Research (PGIMER), Chandigarh, 160 012 India.); Suri V ( Department of Internal Medicine, Postgraduate Institute of Medical Education and Research (PGIMER), Chandigarh, 160 012 India.); Kumari S ( Department of Internal Medicine, Postgraduate Institute of Medical Education and Research (PGIMER), Chandigarh, 160 012 India.); Malhotra P ( Department of Internal Medicine, Postgraduate Institute of Medical Education and Research (PGIMER), Chandigarh, 160 012 India.)
Abstract	Hypereosinophilia is part of a group of complex disorders with multisystem involvement. A 23 year old male was admitted to our centre with bilateral popliteal artery and venous thrombosis and impending gangrene of the left forefoot along with deep venous thrombosis of the right lower extremity. Investigations revealed marked peripheral blood eosinophilia (27,669/µL). Bone marrow showed increased eosinophils & eosinophil precursors and no evidence of a clonal disorder. Skin biopsy from the ulcerated lesions showed small vessel vasculitis with intense eosinophilic infiltration. Investigations to look for secondary causes of hypereosinophilia in the form of Antinuclear Antibody, P-Anti Neutrophil Cytoplasmic Antibody (ANCA) and C-ANCA and FIP1L1-PDGFRA, Bcr-Abl and JAK2V617F mutations were negative. The arterial and venous thrombosis and cutaneous vasculitis were linked to the presence of hypereosinophilic syndrome. The patient's illness responded to high dose corticosteroids leading to complete resolution of symptoms. We reviewed the literature on the lesser known entity of eosinophilic vasculitis and its association with thrombosis.
File Format	HTM / HTML
ISSN	09714502
e-ISSN	09740449
DOI	10.1007/s12288-014-0384-2
Journal	Indian Journal of Hematology and Blood Transfusion
Issue Number	Suppl 1
Volume Number	30
Language	English
Publisher	Springer
Publisher Date	2014-09-01
Publisher Place	India
Access Restriction	Open
Subject Keyword	Discipline Hematology
Content Type	Text
Resource Type	Article
Subject	Hematology

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