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| Content Provider | World Health Organization (WHO)-Global Index Medicus |
|---|---|
| Author | Li, Ming Gong, Shiyan Li, Qing Yuan, Lixia Meng, Fanxing Wang, Rixin |
| Description | Country affiliation: China Author Affiliation: Li M ( School of Marine Sciences, Ningbo University, Ningbo 315211, China.); Gong S ( School of Marine Sciences, Ningbo University, Ningbo 315211, China.); Li Q ( School of Marine Sciences, Ningbo University, Ningbo 315211, China.); Yuan L ( School of Marine Sciences, Ningbo University, Ningbo 315211, China.); Meng F ( School of Marine Sciences, Ningbo University, Ningbo 315211, China.); Wang R ( School of Marine Sciences, Ningbo University, Ningbo 315211, China. Electronic address: wrx_zjou@163.com.) |
| Abstract | A study was carried to test the response of yellow catfish for 28 days under two ammonia concentrations. Weight gain of fish exposure to high and low ammonia abruptly increased at day 3. There were no significant changes in fish physiological indexes and immune responses at different times during 28-day exposure to low ammonia. Fish physiological indexes and immune responses in the treatment of high ammonia were lower than those of fish in the treatment of low ammonia. When fish were exposed to high ammonia, the ammonia concentration in the brain increased by 19-fold on day 1. By comparison, liver ammonia concentration reached its highest level much earlier at hour 12. In spite of a significant increase in brain and liver glutamine concentration, there was no significant change in glutamate level throughout the 28-day period. The total superoxide dismutase (SOD), glutathione peroxidase (GPX) and glutathione reductase (GR) activities in the brain gradually decreased from hour 0 to day 28. Liver SOD, GPX and GR activities reached the highest levels at hour 12, and then gradually decreased. Thiobarbituric acid reactive substance brain and liver content gradually increased throughout the 28-day period. Lysozyme, acid phosphatase and alkaline phosphatase activities in the liver reached exceptionally low levels after day 14. This study indicated that glutamine accumulation in the brain was not the major cause of ammonia poisoning, the toxic reactive oxygen species is not fully counter acted by the antioxidant enzymes and immunosuppression is a process of gradual accumulation of immunosuppressive factors. |
| File Format | HTM / HTML |
| ISSN | 15320456 |
| Journal | Comparative Biochemistry and Physiology Part C: Toxicology & Pharmacology |
| Volume Number | 183-184 |
| Language | English |
| Publisher | Elsevier |
| Publisher Date | 2016-05-01 |
| Publisher Place | United States |
| Access Restriction | One Nation One Subscription (ONOS) |
| Subject Keyword | Discipline Toxicology Discipline Pharmacology |
| Content Type | Text |
| Resource Type | Article |
| Subject | Cell Biology Physiology Health, Toxicology and Mutagenesis Medicine Aquatic Science Biochemistry Toxicology Animal Science and Zoology |
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