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Manipulation or capitulation: virus interactions with autophagy.

Year: 2012 Volume: 14 Issue: 1

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Manipulation or capitulation: virus interactions with autophagy.

Content Provider	World Health Organization (WHO)-Global Index Medicus
Author	Jordan, Tristan X. Randall, Glenn
Description	Country affiliation: United States Author Affiliation: Jordan TX ( Department of Microbiology, The University of Chicago, Chicago, IL 60637, USA.)
Abstract	Autophagy is a homeostatic process that functions to balance cellular metabolism and promote cell survival during stressful conditions by delivering cytoplasmic components for lysosomal degradation and subsequent recycling. During viral infection, autophagy can act as a surveillance mechanism that delivers viral antigens to the endosomal/lysosomal compartments that are enriched in immune sensors. Additionally, activated immune sensors can signal to activate autophagy. To evade this antiviral activity, many viruses elaborate functions to block the autophagy pathway at a variety of steps. Alternatively, some viruses actively subvert autophagy for their own benefit. Manipulated autophagy has been proposed to facilitate nearly every stage of the viral lifecycle in direct and indirect ways. In this review, we synthesize the extensive literature on virus-autophagy interactions, emphasizing the role of autophagy in antiviral immunity and the mechanisms by which viruses subvert autophagy for their own benefit.
File Format	HTM / HTML
ISSN	12864579
e-ISSN	1769714X
DOI	10.1016/j.micinf.2011.09.007
Journal	Microbes and Infection
Issue Number	2
Volume Number	14
Language	English
Publisher	Elsevier
Publisher Date	2012-02-01
Publisher Place	France
Access Restriction	Open
Subject Keyword	Discipline Microbiology Discipline Immunology Autophagy Immunology Host-pathogen Interactions Virus Diseases Viruses Adaptive Immunity Physiology Immunity, Innate Pathology Virology Virus Physiological Phenomena Research Support, N.i.h., Extramural Research Support, Non-u.s. Gov't
Content Type	Text
Resource Type	Article
Subject	Infectious Diseases Immunology Microbiology

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