NDLI: Deficient inhibition of return in chronic but not first-episode patients with schizophrenia.

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Abnormality of cerebral perfusion in the posterior cingulate gyrus of a refractory patient with schizophrenia and minocycline treatment.

Lower mRNA BDNF expression in lymphocytes: endophenotype or epiphenomenon for major depression?

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Serotonin transporter gene polymorphisms and treatment-resistant depression.

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Deficient inhibition of return in chronic but not first-episode patients with schizophrenia.

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Pharacogenetic effects of dopamine transporter gene polymorphisms on response to chlorpromazine and clozapine and on extrapyramidal syndrome in schizophrenia.

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Hepatic drug metabolizing profile of Flinders Sensitive Line rat model of depression.

Effect of quetiapine-XR on irritable bowel syndrome.

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Deficient inhibition of return in chronic but not first-episode patients with schizophrenia.

Content Provider	World Health Organization (WHO)-Global Index Medicus
Author	Liu, Dengtang Fan, Xiaoduo Wang, Yan Yang, Zhiliang Zhuo, Kaiming Song, Zhenhua Wu, Yan Li, Chunbo Wang, Jijun Xu, Yifeng
Description	Country affiliation: China Author Affiliation: Liu D ( Department of Psychiatry, Shanghai Mental Health Center, Shanghai Jiao Tong University School of Medicine, Shanghai 200030, China.)
Abstract	BACKGROUND: Inhibition of return (IOR) has been tested in patients with schizophrenia with contradictory results. Some studies indicated that patients with schizophrenia have normal levels of IOR; however, other studies reported delayed or blunted IOR. Inconsistency in findings might be due to differences across studies in relevant aspects associated with disease, such as heterogeneity of the disorder, different medications, onset and severity of the illness. The present study was to explore different patterns of IOR in antipsychotic medication free first-episode schizophrenia and chronic schizophrenia. METHODS: Forty two patients with first-episode schizophrenia, 44 patients with chronic schizophrenia, and 38 healthy controls were included in the study. All subjects went through a covert orienting of attention task with seven stimulus onset asynchrony (SOA) intervals (400 ms, 500 ms, 600 ms, 700 ms, 800 ms, 1200 ms and 1500 ms). RESULTS: Compared with healthy controls, the magnitude and onset of IOR in first-episode patients with schizophrenia were intact. However, in patients with chronic schizophrenia, there was an attenuated cuing effect especially at SOA 700 ms; in addition, there was a robust IOR until at SOAs 800 ms or above. Moreover, the illness duration and the number of psychotic episodes were significantly correlated with the validity effect at SOAs 400 ms and 600 ms. CONCLUSION: Our study suggests that deficient IOR presents in chronic but not in first-episode patients with schizophrenia. IOR deficit in schizophrenia may begin during the course of illness and deteriorate over the course of illness. Our findings are consistent with the neurodegenerative model of schizophrenia.
File Format	HTM / HTML
ISSN	02785846
Issue Number	6
Volume Number	34
e-ISSN	18784216
Journal	Progress in Neuro-Psychopharmacology and Biological Psychiatry
Language	English
Publisher	Elsevier
Publisher Date	2010-08-16
Publisher Place	Great Britain (UK)
Access Restriction	One Nation One Subscription (ONOS)
Subject Keyword	Discipline Psychiatry Attention Physiology Cognition Inhibition (psychology) Schizophrenia Physiopathology Adult Analysis Of Variance Cues Female Humans Male Neuropsychological Tests Reaction Time Journal Article Research Support, Non-u.s. Gov't
Content Type	Text
Resource Type	Article
Subject	Biological Psychiatry Pharmacology

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