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| Content Provider | Springer Nature Link |
|---|---|
| Author | Galenko Yaroshevskii, V. P. Galka, V. V. Stoyalova, O. N. Agadzhava, A. V. Lapina, I. V. Gaivoronskaya, T. V. Popkova, L. V. Churilova, I. V. Gorelashvili, A. S. Antelava, N. A. Berberashvili, T. M. Sukoyan, G. V. |
| Copyright Year | 2006 |
| Abstract | Antinecrotic activity of SOD was studied in rats with experimental skin ischemia. Treatment with SOD increased activity of endogenous SOD in skin homogenates (by 70 and 26% compared to the ischemic and intact skin, respectively). However, the rate of superoxide anion generation remained unchanged after SOD treatment. Creatine phosphate content and NAD/NADH redox potential increased by 16 and 21%, respectively, on day 3 after SOD administration. The increase in functional activity of the energy supply system and rise in the reserve capacity of the antioxidant protection system contribute to inhibition of lactate dehydrogenase and creatine phosphokinase and decrease in the cytolysis index under the influence of SOD. Our results indicate that SOD produces the antinecrotic effect and holds much promise for the therapy of skin ischemia. |
| Starting Page | 447 |
| Ending Page | 449 |
| Page Count | 3 |
| File Format | |
| ISSN | 00074888 |
| Journal | Bulletin of Experimental Biology and Medicine |
| Volume Number | 142 |
| Issue Number | 4 |
| e-ISSN | 15738221 |
| Language | English |
| Publisher | Kluwer Academic Publishers-Consultants Bureau |
| Publisher Date | 2006-01-01 |
| Publisher Place | New York |
| Access Restriction | One Nation One Subscription (ONOS) |
| Subject Keyword | superoxide dismutase skin ischemia necrotic changes Biomedicine general Laboratory Medicine Internal Medicine Pathology Cell Biology |
| Content Type | Text |
| Resource Type | Article |
| Subject | Medicine Biochemistry, Genetics and Molecular Biology |
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