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Obsessive compulsive disorder and basal ganglia dysfunction.
| Content Provider | Semantic Scholar |
|---|---|
| Author | Rapoport, Judith L. |
| Copyright Year | 1990 |
| Abstract | There is increasing evidence that selective basal ganglia dysfunction underlies obsessive-compulsive disorder (OCD). Recent neuroanatomical, neuropharmacological, and behavioural studies indicate a complex perceptual and cognitive role for the basal ganglia in addition to their more well-accepted motor functions. In addition, obsessive-compulsive symptoms in certain syndromes and in individual cases with basal ganglia disease, response to psychosurgery, and new antiobsessional drugs and recent brain-imaging studies all support such a model. OCD may represent the inappropriate triggering of genetically stored and learned behaviours by the striatum as the primary cause of OCD. |
| File Format | PDF HTM / HTML |
| DOI | 10.1017/S0033291700016962 |
| Alternate Webpage(s) | https://www.cambridge.org/core/services/aop-cambridge-core/content/view/957400DA2016A2A3A0DCB6DE8E6C2265/S0033291700016962a.pdf/obsessive_compulsive_disorder_and_basal_ganglia_dysfunction1.pdf |
| PubMed reference number | 2236357 |
| Alternate Webpage(s) | https://doi.org/10.1017/S0033291700016962 |
| Journal | Medline |
| Volume Number | 20 |
| Issue Number | 3 |
| Journal | Psychological medicine |
| Language | English |
| Access Restriction | Open |
| Content Type | Text |
| Resource Type | Article |
