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Significance of the diagnostic Q wave of myocardial infarction.
| Content Provider | Semantic Scholar |
|---|---|
| Author | Horan, Leo G. Flowers, Nancy C. Johnson, Jennifer C. |
| Copyright Year | 1971 |
| Abstract | SUMMARY Correlation between the QRS complex and postmortem ventricular anatomy was made in 1184 instances of normal conduction: (1) Mechanical reliance on the sheer presence or absence of a Q wave greater than 0.03 sec in duration led to "correct" diagnosis of infarction or not in 79% of the series. (2) With normal conduction, abnormal Q waves isolated to either the anteroseptal (Vl-V4) or inferior (II, Ill, aVF) electrocardiographic zones were frequently false (46%). However, abnormal Q waves restricted to the lateral zone (V5-V6) or in a combination of more than one electrocardiographic zone, were rarely false predictors of the presence of infarction (4%). (3) Classical localization of infarction with normal conduction was statistically relatively reliable as compared with bundle-branch block. The increased frequency of the anatomic pattern of lateral basal infarction in association with normal QRS complexes (but known infarction) suggests relative "electrical silence" of the latero-basal left ventricle in abnormal Q-wave genesis. (4) Lesions confined to a given anatomic location in the left ventricle tended to place particular emphasis and limits on the spectrum of electrocardiographic expression but did not yield a uniform single pattern of Q-wave distribution. Abnormal Q wave Clinicopatholog Electrocardiogram T-I HE ASSOCIATION between an abnormally wide Q wave and myocardial infarction has become so firmly entrenched in the mind of the electrocardiographer as to approach dogma. Monumental work of Feni-chel and Kugell,' Wilson and coworkers,2 and Myers and coworkers3-9 established the theoretical and empirical basis for association 428 tic correlation Myocardial scar between myocardial necrosis and Q-wave abnormality. All of these workers recognized that the association was high but not absolute. In a classic experiment, Bayley and LaDue'0 demonstrated appearance of an abnormal Q wave within minutes after transient coronary occlusion in the experimental animal-a finding later confirmed by many and shown to be reversible, thus providing evidence that the new Q wave is not synonymous with myocar-dial necrosis. Simonson,"1 in examining the statistical basis of clinical and electrocardio-graphic associations, reminded us that the arbitrary division between populations of normal and abnormal (with respect to myo-cardial infarction) by the width of the Q wave in lead III is at best a crude one. Our original interest in critical examination of the relationship between the presence of an |
| Starting Page | 1 |
| Ending Page | 5 |
| Page Count | 5 |
| File Format | PDF HTM / HTML |
| Alternate Webpage(s) | http://circ.ahajournals.org/content/circulationaha/43/3/428.full.pdf |
| Alternate Webpage(s) | http://circ.ahajournals.org/content/circulationaha/43/3/428.full.pdf?download=true |
| PubMed reference number | 5544988v1 |
| Volume Number | 43 |
| Issue Number | 3 |
| Journal | Circulation |
| Language | English |
| Access Restriction | Open |
| Subject Keyword | Anatomic Site Anatomic structures Animals, Laboratory Autopsy Behavioral tic Bundle-Branch Block Cardiomyopathies Coronary Occlusion Dial Device Component Electrocardiography Heart Ventricle Left ventricular structure Massive Hepatic Necrosis Mental association Myocardial Infarction Pichia sp. feni 108 Seventy Nine width |
| Content Type | Text |
| Resource Type | Article |
