NDLI: Model for chronic overexpression of NGF challenges old paradigms: focus on "overexpression of NGF in mouse urothelium leads to neuronal hyperinnervation, pelvic sensitivity, and changes in urinary bladder function".

Content Provider	Semantic Scholar
Author	Allen, Arielle M. Saban, Ricardo
Copyright Year	2010
Abstract	nerve growth factor (ngf) has been suggested to play a role in bladder pain by mediating inflammation ([12][1]) and functional changes in sensory and sympathetic neurons innervating the urinary bladder ([5][2]). Clinically, diseases such as interstitial cystitis/painful bladder syndrome (IC/PBS)
Starting Page	33
Ending Page	33
Page Count	1
File Format	PDF HTM / HTML
DOI	10.1152/ajpregu.00001.2010
Alternate Webpage(s)	http://ajpregu.physiology.org/content/ajpregu/298/3/r532.full.pdf
Alternate Webpage(s)	http://ajpregu.physiology.org/content/ajpregu/early/2010/01/20/ajpregu.00001.2010.full.pdf
PubMed reference number	20089712
Alternate Webpage(s)	https://doi.org/10.1152/ajpregu.00001.2010
Journal	Medline
Volume Number	298
Issue Number	3
Journal	American journal of physiology. Regulatory, integrative and comparative physiology
Language	English
Access Restriction	Open
Content Type	Text
Resource Type	Article

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Model for chronic overexpression of NGF challenges old paradigms: focus on "overexpression of NGF in mouse urothelium leads to neuronal hyperinnervation, pelvic sensitivity, and changes in urinary bladder function".