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Is Emilin-1 a molecular link contributing to the extension of thoracic aortic aneurysm dissection and increasing the magnitude of the associated hypertension
| Content Provider | Semantic Scholar |
|---|---|
| Copyright Year | 2016 |
| Abstract | Patients with acute aortic dissection, the dominant condition in acute aortic syndromes, have a high mortality [1]. Within types of thoracic aortic dissection, the more extensive the TAD the worse the prognosis [2]. Thus, factors influencing the extent of the TAD are important considerations. The initial presentation of acute aortic dissection is severe chest pain and hypertension. The presence of hypertension is a key element of thoracic aortic dissection (TAD) [3]. The hypertension in acute aortic syndromes is often severe and difficult to treat. From another perspective, a significant proportion of patients presenting to the emergency department with hypertensive crisis with severe blood pressure elevations have an aortic dissection [4]. |
| File Format | PDF HTM / HTML |
| Alternate Webpage(s) | https://www.oatext.com/pdf/IMM-3-254.pdf |
| Language | English |
| Access Restriction | Open |
| Subject Keyword | Acute inflammatory demyelinating polyneuropathy Aortic Aneurysm, Thoracic Aortic Valve Insufficiency Chest Pain Contribution Dissecting aortic aneurysm Dissection of aorta Dissection problem Hypertensive crisis Hypertensive disease Patients Syndrome |
| Content Type | Text |
| Resource Type | Article |
