NDLI: Loss of matrilin 1 does not exacerbate the skeletal phenotype in a mouse model of multiple epiphyseal dysplasia caused by a Matn3 V194D mutation

Content Provider	Scilit
Author	Bell, Peter A. Pirog, Katarzyna Fresquet, Maryline Thornton, David Boot-Handford, Raymond Briggs, Michael D.
Copyright Year	2012
Description	Journal: Arthritis & Rheumatism
Related Links	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3374853/pdf
Ending Page	1539
Page Count	11
Starting Page	1529
ISSN	00043591
e-ISSN	15290131
DOI	10.1002/art.33486
Journal	Arthritis & Rheumatism
Issue Number	5
Volume Number	64
Language	English
Publisher	Wiley-Blackwell
Publisher Date	2012-04-26
Access Restriction	Open
Subject Keyword	Journal: Arthritis & Rheumatism Biochemistry and Molecular Biology Extracellular Matrix Mutant Protein
Content Type	Text
Resource Type	Article
Subject	Immunology and Allergy Immunology Pharmacology (medical) Rheumatology

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Loss of matrilin 1 does not exacerbate the skeletal phenotype in a mouse model of multiple epiphyseal dysplasia caused by a Matn3 V194D mutation