NDLI: Anti-Idiotype Antibodies In Myasthenia Gravis

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Anti-Idiotype Antibodies In Myasthenia Gravis

Content Provider	Scilit
Author	Lefvert, A. K.
Copyright Year	2018
Description	The network theory as hypothesized by N. K. Jerne assumes the ability of anti-idiotype (anti-Id) antibodies both to suppress and to enhance specific immune responses. The receptor antibodies in the human disease are polyclonal. The antibodies differ with regard to binding sites on the receptor, isoelectric point, and reactivity with anti-Id antibodies. There is abundant evidence that all antibodies which bind to the acetylcholine receptor can disturb the neuromuscular function. Abnormal immune regulation has been suggested to contribute to the development and perpetuation of autoimmune disorders, and in human autoimmune diseases, autologous anti-Id have been demonstrated in myasthenia gravis, in systemic lupus erythematosus, in insulin-dependent diabetes, and in autoimmune disorders of the thyroid gland. There are two instances in which one can talk about a transient myasthenia gravis syndrome: one is neonatal myasthenia and the other is the penicillamine-induced disease. Book Name: Biological Applications of Anti-Idiotypes
Related Links	https://api.taylorfrancis.com/content/chapters/edit/download?identifierName=doi&identifierValue=10.1201/9781351070225-5&type=chapterpdf
Ending Page	90
Page Count	22
Starting Page	69
DOI	10.1201/9781351070225-5
Language	English
Publisher	Informa UK Limited
Publisher Date	2018-01-18
Access Restriction	Open
Subject Keyword	Book Name: Biological Applications of Anti-idiotypes Imaging Science Otorhinolaryngology Antibodies Insulin Myasthenia Gravis Autoimmune Idiotype Diabetes Function
Content Type	Text
Resource Type	Chapter

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