NDLI: The interaction between human enteroviruses and type I IFN signaling pathway

Please wait, while we are loading the content...

The interaction between human enteroviruses and type I IFN signaling pathway

Content Provider	Scilit
Author	Lu, Jing Yi, Lina Ke, Changwen Zhang, Yonghui Liu, Ren Chen, Jinfei Kung, Hsiang-Fu He, Ming-Liang
Copyright Year	2013
Description	Human enteroviruses (HEV), very common and important human pathogens, cause infections in diverse ways. Recently, the large epidemic of HFMD caused by HEV infection became a growing threat to public health in China. As the first line of immune response, the type I interferon (IFN-α/β) pathway plays an essential role in antiviral infection, particularly in limiting both the early and late stages of infection. Because of co-evolution with the host, the viruses have evolved multiple strategies to evade or subvert the host immunity to ensure their survival. In this paper, we systematically reviewed and summarized the interaction between HEV infections and host type I IFN responses. We firstly described the recent findings of HEV recognition and IFN induction, specifically on host pattern-recognition receptors (PRRs) in HEV infection. Then we discussed the antiviral effect of IFN in HEV infection. Finally, we timely summarized the mechanisms of HEV to circumvent the IFN responses. Clarification of the complexity in this battle may provide us new strategies for prevention and antiviral treatment.
Related Links	https://www.tandfonline.com/doi/pdf/10.3109/1040841X.2013.813903
Ending Page	207
Page Count	7
Starting Page	201
ISSN	1040841X
e-ISSN	15497828
DOI	10.3109/1040841x.2013.813903
Journal	Critical reviews in microbiology
Issue Number	2
Volume Number	41
Language	English
Publisher	Informa UK Limited
Publisher Date	2013-07-16
Access Restriction	Open
Subject Keyword	Journal: Critical Reviews in Microbiology Virology Antiviral Host-virus Interaction Human Enterovirus Interferon Respons Viral Replication
Content Type	Text
Resource Type	Article
Subject	Medicine Microbiology Applied Microbiology and Biotechnology

Sl.	Authority	Responsibilities	Communication Details
1	Ministry of Education (GoI), Department of Higher Education	Sanctioning Authority	https://www.education.gov.in/ict-initiatives
2	Indian Institute of Technology Kharagpur	Host Institute of the Project: The host institute of the project is responsible for providing infrastructure support and hosting the project	https://www.iitkgp.ac.in
3	National Digital Library of India Office, Indian Institute of Technology Kharagpur	The administrative and infrastructural headquarters of the project	Dr. B. Sutradhar bsutra@ndl.gov.in
4	Project PI / Joint PI	Principal Investigator and Joint Principal Investigators of the project	Dr. B. Sutradhar bsutra@ndl.gov.in Prof. Saswat Chakrabarti will be added soon
5	Website/Portal (Helpdesk)	Queries regarding NDLI and its services	support@ndl.gov.in
6	Contents and Copyright Issues	Queries related to content curation and copyright issues	content@ndl.gov.in
7	National Digital Library of India Club (NDLI Club)	Queries related to NDLI Club formation, support, user awareness program, seminar/symposium, collaboration, social media, promotion, and outreach	clubsupport@ndl.gov.in
8	Digital Preservation Centre (DPC)	Assistance with digitizing and archiving copyright-free printed books	dpc@ndl.gov.in
9	IDR Setup or Support	Queries related to establishment and support of Institutional Digital Repository (IDR) and IDR workshops	idr@ndl.gov.in

The virology and developments toward control of human enterovirus 71

ATM supports gammaherpesvirus replication by attenuating type I interferon pathway

Interferon-Induced Protein 44 Interacts with Cellular FK506-Binding Protein 5, Negatively Regulates Host Antiviral Responses, and Supports Virus Replication.

Porcine Respirovirus 1 Suppresses Host Type I Interferon Production and the JAK-STAT Signaling Pathway.

Potent inhibition of human enterovirus 71 replication by type I interferon subtypes.

IFITM2 Presents Antiviral Response through Enhancing Type I IFN Signaling Pathway.

Interferon-Induced Protein 44 Interacts with Cellular FK506-Binding Protein 5, Negatively Regulates Host Antiviral Responses, and Supports Virus Replication

Echovirus induces autophagy to promote viral replication via regulating mTOR/ULK1 signaling pathway

Zinc Finger CCCH-Type Antiviral Protein 1 Restricts the Viral Replication by Positively Regulating Type I Interferon Response

The interaction between human enteroviruses and type I IFN signaling pathway

Similar Documents

The virology and developments toward control of human enterovirus 71

ATM supports gammaherpesvirus replication by attenuating type I interferon pathway

Interferon-Induced Protein 44 Interacts with Cellular FK506-Binding Protein 5, Negatively Regulates Host Antiviral Responses, and Supports Virus Replication.

Porcine Respirovirus 1 Suppresses Host Type I Interferon Production and the JAK-STAT Signaling Pathway.

Potent inhibition of human enterovirus 71 replication by type I interferon subtypes.

IFITM2 Presents Antiviral Response through Enhancing Type I IFN Signaling Pathway.

Interferon-Induced Protein 44 Interacts with Cellular FK506-Binding Protein 5, Negatively Regulates Host Antiviral Responses, and Supports Virus Replication

Echovirus induces autophagy to promote viral replication via regulating mTOR/ULK1 signaling pathway

Zinc Finger CCCH-Type Antiviral Protein 1 Restricts the Viral Replication by Positively Regulating Type I Interferon Response

The interaction between human enteroviruses and type I IFN signaling pathway