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| Content Provider | frontiers |
|---|---|
| Author | Justulin, Luis A. Zambrano, Elena Ong, Thomas P. Ozanne, Susan E. |
| Abstract | Over the past decades, the Developmental Origins of Health and Disease (DOHaD) has been consolidated as a concept asserting the causal effects of early life exposure to environmental stressors (including malnutrition, pollutants, and stress) and the global increase in non-communicable chronic diseases observed in modern society (Gluckman et al., 2010). Although multiple mechanisms have been proposed to underlie developmental programming, epigenetic processes (including DNA methylation, histone post-translational modifications, and dysregulated non-coding RNA expression) have been described as a key mechanistic framework contributing to the non-genomic heritable increase in risk disease (Treviño et al., 2020). The articles published in this Research addressed several aspects of how early life exposure to different adverse conditions may influence health and diseases throughout the life span. Sinzato et al. (2022) demonstrated, the negative impact of diabetes combined with lifelong high-fat diet consumption on reproductive parameters in dams, while Garcia-Santillan et al. (2022), Chavira-Suárez et al. (2022), and Simino et al. (2022) explored, respectively, the role of maternal consumption of obesogenic diet on the placental expression of nutrient transporters, methylation status in umbilical cords, and miRNA expression profile in offspring liver regeneration after partial hepatectomy. The influence of parental high-fat high-sugar diet intake on epigenetic markers and the re... |
| ISSN | 2296634X |
| DOI | 10.3389/fcell.2023.1139283 |
| Volume Number | 11 |
| Journal | Frontiers in Cell and Developmental Biology |
| Language | English |
| Publisher Date | 2023-02-10 |
| Access Restriction | Open |
| Subject Keyword | Developmental plasticity DOHAD Epigenetics Aging Adult disease |
| Content Type | Text |
| Resource Type | Article |
| Subject | Cell Biology Developmental Biology |
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